SLFN11 Promotes CDT1 Degradation by CUL4 in Response to Replicative DNA Damage, While its Absence Leads to Synthetic Lethality With ATR/CHK1 Inhibitors

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SLFN11 Promotes CDT1 Degradation by CUL4 in Response to Replicative DNA Damage, While its Absence Leads to Synthetic Lethality With ATR/CHK1 Inhibitors

Functional Genomics Lab https://pubmed.ncbi.nlm.nih.gov/33536335/ 2021-02-09T00:00:00Z Proceedings of the National Academy of Sciences of the United States of America

Citation Text

SLFN11 Promotes CDT1 Degradation by CUL4 in Response to Replicative DNA Damage, While its Absence Leads to Synthetic Lethality With ATR/CHK1 Inhibitors